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12 articles
Type 1 diabetes may not begin as a sudden immune attack, but as a slow beta-cell failure in which viral stress and genetic context reshape the cellsâ defenses.
The Geneva experiment does not end insulin tomorrow, but it shows how a bioartificial pancreas could survive in the body long enough to become a serious therapeutic platform.
New research suggests a daily GLP-1 pill could help maintain results achieved with semaglutide or tirzepatide.
Swedish researchers developed a more reliable way to make insulin-producing cells that restored glucose control in mice.
A new preclinical study changes an old assumption: for mRNA vaccines, the key is not only which antigen is delivered, but where the body expresses it.
WEHIâs Nature paper reveals ubiquitin tags can bind glycogen, upending 70 years of metabolic dogma.
Anglia Ruskin University led the research, which involved collaboration with Cranfield University, the University of Portsmouth, and Intelligent Omics Ltd.
A Nature Metabolism study reveals metformin activates the AMPK pathway via the PEN2 protein.
A breakthrough noninvasive method measures beta cell mass in living patientsâpotentially rewriting type 1 diabetes treatment strategies.
A father and daughter with no lifestyle risk factors led Mayo Clinic to uncover a rare genetic cause of fatty liver disease.
LRG1, a protein previously linked to blood vessel formation, now stands accused of starving retinal cells in diabetic mice by constricting their microvasculature.
East Asian men with a missing Y chromosome in their pancreatic cells face 30% higher diabetes oddsâ*but only if their genetic risk is otherwise low*, per a *Nature Medicine* analysis of 200,000+ genomes.
